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Imagine a packet of soup with a bowl of soup on the front of the packaging. How worried should we be by the rise of food porn?

Nutritional ad

Received Apr 3; Accepted Sep The use, distribution or reproduction in other forums is permitted, provided the original author s or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

This article has been cited by other articles in PMC. Abstract A nutritional approach to prevent, slow, or halt the progression of disease is a promising strategy that has been widely investigated. Modifiable, environmental causes of AD include potential metabolic derangements caused by dietary insufficiency and or excess that may be corrected by nutritional supplementation and or dietary modification.

Many nutritional supplements contain a myriad of health promoting constituents anti-oxidants, vitamins, trace minerals, flavonoids, lipids, …etc. Nutritional modifications have the advantage of being cost effective, easy to implement, socially acceptable and generally safe and devoid of significant adverse events in most cases.

Many nutritional interventions have been studied and continue to be evaluated in hopes of finding a successful agent, combination of agents, or dietary modifications that can be used for the prevention and or treatment of AD. The current review focuses on several key nutritional compounds and dietary modifications that have been studied in humans, and further discusses the rationale underlying their potential utility for the prevention and treatment of AD.

First described by the German psychiatrist and neuropathologist, Alois Alzheimer, inthe eponymous syndrome is recognized worldwide as a major cause of morbidity and mortality in the aging population contributing to a major burden on healthcare systems Brookmeyer et al.

Amyloid plaque deposition in the brain, the development of neurofibrillary pathology, neuronal loss and dysfunction of neuroanatomic systems, including those affecting cholinergic transmission, are among the commonly described findings in persons with AD Jicha and Carr, In addition to these core pathological features of AD, much evidence has accumulated that increased oxidative stress, defects in mitochondrial dysfunction and cellular energy production, and chronic inflammatory mechanisms contribute to the degenerative cascade in this complex disease Rao and Balachandran, ; Cunnane et al.

A cure for the condition is not known and current treatment approaches include maximizing transmission of acetylcholine and other neurotransmitters while also using other medications and multidisciplinary strategies to treat associated comorbidities focused on improving quality of life and alleviating symptom burden Jicha and Carr, A recent NIH scientific roundtable concluded that there is no definitive evidence for benefit of any preventive strategy for AD, however, this working group also concluded that preventative strategies focused on modifying environmental risk factors deserves further scientific exploration and study Daviglus et al.

Researchers in the field of AD were also underrepresented in the consensus group leading to further debate as to the validity of the consensus statement.

As such it is clear that the field is far from consensus on the debate of whether viable interventions for the prevention of AD exist. While the search for a cure remains elusive, there is much hope that preventative strategies, such as dietary modification and nutritional supplementation, may reduce the global burden of AD.Get nutrition advice from Publix.

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Junk food advertising, which is linked to increased child weight and obesity, sharpens this ethical issue, compounding it with health concerns. From three years of age, children recognise more.

Nutritional ad

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